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Inflachromene ameliorates Parkinson's disease by targeting Nrf2-binding Keap1

  • Junhyeong Yim
  • , Yoon Soo Hwang
  • , Jae Jin Lee
  • , Ju Hee Kim
  • , Jeong Yeob Baek
  • , Jaeyeong Jeong
  • , Young Il Choi
  • , Byung Kwan Jin
  • , Seung Bum Park

Research output: Contribution to journal › Article › peer-review

6 Citations (Scopus)

Abstract

Parkinson's disease (PD) is the most common neurodegenerative disease characterized by movement disorder. Despite current therapeutic efforts, PD progression and the loss of dopaminergic neurons in the substantia nigra remain challenging to prevent due to the complex and unclear molecular mechanism involved. We adopted a phenotype-based drug screening approach with neuronal cells to overcome these limitations. In this study, we successfully identified a small molecule with a promising therapeutic effect for PD treatment, called inflachromene (ICM), through our phenotypic screening strategy. Subsequent target identification using fluorescence difference in two-dimensional gel electrophoresis (FITGE) revealed that ICM ameliorates PD by targeting a specific form of Keap1. This interaction led to upregulating various antioxidants, including HO-1, NQO1, and glutathione, ultimately alleviating PD symptoms. Furthermore, ICM exhibited remarkable efficacy in inhibiting the loss of dopaminergic neurons and the activation of astrocytes and microglia, which are critical factors in PD pathology. Our findings suggest that the phenotypic approach employed in this study identified that ICM has potential for PD treatment, offering new hope for more effective therapeutic interventions in the future.

Original languageEnglish
Pages (from-to)3588-3595
Number of pages8
JournalChemical Science
Volume15
Issue number10
DOIs
Publication statusPublished - 2 Feb 2024

Bibliographical note

Publisher Copyright:
© 2024 The Royal Society of Chemistry.

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