Protective role of intestinal bacterial metabolism against baicalin-induced toxicity in HepG2 cell cultures

Tilak Khanal, Hyung Gyun Kim, Jae Ho Choi, Bong Hwan Park, Minh Truong Do, Mi Jeong Kang, Hee Kyung Yeo, Dong Hyun Kim, Wonku Kang, Tae Cheon Jeong, Hye Gwang Jeong

Research output: Contribution to journalArticlepeer-review

22 Citations (Scopus)

Abstract

Baicalin, a glycoside present in Scutellaria baicalensis Georgi, is metabolized to its aglycone, baicalein, in intestine. In the present study, possible role of metabolism of baicalin by intestinal bacteria to baicalein in baicalin-induced toxicity was investigated in HepG2 cell cultures. As an intestinal bacterial metabolic system for baicalin, human fecal preparation containing intestinal microflora (fecalase) was employed. Initially, when cytotoxic effects of baicalin and baicalein were compared, baicalin was more cytotoxic than baicalein in HepG2 cells. When baicalin was incubated with fecalase, it was metabolized to baicalein. In addition, baicalin-incubated with fecalase reduced cytotoxicity of HepG2 cells in a concentration-dependent manner. Moreover, baicalin-incubated with fecalase significantly caused an increase in Bcl-2 expression together with a decrease in Bax expression and cleaved Caspase-3. Furthermore, anti-apoptotic effect by the incubation of baicalin with fecalase was also confirmed by the terminal deoxynucleotidyltransferase-mediated dUTP-biotin nick-end labeling assay. Taken all together, the findings suggested that metabolism of baicalin by human fecalase to baicalein might have protective effects against baicalin-induced toxicity in HepG2 cells.

Original languageEnglish
Pages (from-to)363-371
Number of pages9
JournalJournal of Toxicological Sciences
Volume37
Issue number2
DOIs
Publication statusPublished - Apr 2012

Keywords

  • Baicalein
  • Baicalin
  • Cytotoxicity
  • Intestinal microflora
  • Metabolism
  • Protection

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